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One major mediator of this event is transforming growth factor-beta (TGF-), which is secreted by the tumor stroma and participates in a paracrine signaling loop with tumor cells [19]

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This decline has led some researchers to speculate that reduced GHK signaling could contribute to age-related impairments in: wound healing tissue repair collagen production While this hypothesis remains under investigation, it helps explain why GHK-Cu supplementationparticularly through topical formulationshas become a focus of dermatologic research

Then, we specifically focus on the signaling pathway mediated by hepatocyte growth factor (HGF) and its receptor c-Met, which play an important role during physiological processes and were been associated with tumorigenesis

It is not a growth hormone or a hormone in the classical sense