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One further general requirement for homeostatic mechanism(s) is the time scale of the induction of synaptic changes

Data are from the on-treatment observation period (during treatment with trial product [any dose of trial medication administered within the previous 49 days (that is, any period of temporary treatment interruption with trial product was excluded))

This damage is exacerbated by the fact that the heart has a high metabolic rate and oxygen demand, making it particularly vulnerable to oxidative injury [2]

One major obstacle is the difficulty in translating animal model results into human clinical settings due to species-specific variations in drug metabolism, pharmacokinetics, and the complexity of arrhythmias in human patients

Open Heart 8 , e001755 (2021)