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Mitochondrial ROS production and oxidative mtDNA damage is increased during reoxygenation in the GPX1 knockout mice, in addition to structural abnormalities in cardiac mitochondria and myocytes, suggesting GPX1 may play an important role in protecting cardiac mitochondria from reoxygenation damage in vivo

Z.FischerR

In this approach, CAT-containing samples are introduced to defined H 2 O 2 concentrations in phosphate buffer (pH 7.0), with absorbance decline rates proportional to enzymatic activity

Levels of 0.40% or higher are potentially fatal, with risks including coma and death due to respiratory arrest

Intended for research use only in qualified laboratory settings