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GSH as oxygen consumption substrate of mitochondria GSH do not change the basic O 2 consumption of rat liver mitochondria at pH 7.0 and 1 mM EDTA without added Mg 2+ or Ca 2+

Khan KA, Kerbel RS

The regulator requires satisfactory responses from this third-party manufacturer before the CRL can be resolved, according to Camurus

We measured the response to ADP stimulation in young and old muscle mitochondria with ELAM treatment, in vivo heart and muscle function, and compared protein abundance, phosphorylation, and S-glutathionylation of ADP/ATP pathway proteins

CLL/N M cells were characterized by elevated ECAR compared to CLL/N WT together with a low lactate production, suggesting an enhanced glycolytic flux directed toward mitochondrial metabolism (Fig