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This is achieved through the promotion of NADH oxidation and inhibition of mitochondrial permeability transition, both of which are crucial factors for maintaining cellular energy and survival [19]

Utilizing this, various pharmacological inhibitors and non-coding RNAs can be delivered that could inhibit the biogenesis of LDs or reduce their sizes, reversing the excess lipid-related imbalance in neurons

These consolidated sections reduce redundancy and clarify the interrelationship between oxidative stress, mitochondrial damage, and apoptosis while preserving mechanistic depth and scientific accuracy

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A pulse radiolysis study of the reaction of hydrated electrons with N formylkynurenine and related compounds: electron transfer reactions with nucleic acid components