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Description

Impacts neurotransmitter metabolism and nitric oxide regulation

A previous study supported this, showing that treatment of SLC1A5 -knockdown HNSCC cell lines with V-9302 led to marked inhibition of glutamine metabolism, thereby suppressing growth and proliferation both in vitro and in vivo 97

Transcriptional programming of lipid and amino acid metabolism by the skeletal muscle circadian clock

On the other hand, cyclization of the 3-chloroacetamide restored activity (cluster-5)

These findings suggest that NHPN-1010 could be a beneficial agent for the treatment of NIT through reversal of cochlear synapthopathy in the IHC area and suppression of neuronal hyperactivity in the CAS