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iron-mediated programmed cell death that is characterized by oxidative cell death resulting from iron-dependent accumulation of lipid reactive species

This situation would lead to a self-perpetuating cycle, in which the free radicals generated would induce GSH depletion, thus increasing oxidative stress that would reduce antioxidant enzyme levels, which would further reduce GSH synthesis

10.1016/j.chembiol.2016.12.007 23 KerinsM

Under normal physiological conditions, about 3% of cytosolic glucose is processed via the polyol pathway, however, at higher concentrations of glucose, about 30% of the glucose enters the polyol pathway which makes it important in disposing of the glucose molecules and subsequent conversion to sorbitol [128]

By 8 weeks, M B cells frequency dropped significantly, indicating that Gclc -deficient T1 B cells limit M B cells development and that Gclc is crucial for the transition from T1 to T2 B cells