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Moreover, the dynamic communication of L-Phe at its predicted binding site in the hinge region with the Ca 2+ binding sites not only influences the adjacent Ca 2+ binding site 1, but also globally enhances cooperative activation of the receptor in response to alterations in extracellular Ca 2+

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Conclusion and future perspectives The role of ROS in tumor immunity is governed by a fundamental paradox: ROS are indispensable for inducing ICD and activating cytotoxic lymphocytes, yet excessive or chronic ROS drive immune exhaustion and sustain an immunosuppressive TME