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Prolonged metabolic stress and redox imbalance alter substrate preference, hinder pyruvate dehydrogenase activity, and decrease the efficacy of oxidative phosphorylation, ultimately resulting in energetic insufficiency (3, 41)

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When medical ozone contacts blood components, it creates controlled oxidative exposure that paradoxically reduces chronic oxidative stress over time by upregulating endogenous antioxidant systems: glutathione, superoxide dismutase, and catalase production increases in response