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These findings suggest a potential mechanism for the beneficial effects of CB 1 receptor antagonism at the behavioral level

However, tumor cells often downregulate HLA-I and allow effector NK cells to be activated by immunoreceptor tyrosine-based activation motifs (ITAMs) in intracellular domains, resulting in the release of cytokines, perforins and granzyme B and leading to tumor cell death

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Lowe, Senescence-induced vascular remodeling creates therapeutic vulnerabilities in pancreas cancer