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The molecular mechanisms of ferroptosis in diabetic complications involve multiple proteins, including acyl-CoA synthetase long chain family member 4 (ACSL4), high-mobility group box-1 (HMGB1), hypoxia-inducible factor-1 (HIF-1), heme oxygenase-1 (HO-1), tripartite motif containing 46 (TRIM46), circ-PSEN1, NCOA4 (68), and Nox2, and negative genes, Gpx4 , Nrf2 , xCT , adenosine monophosphate-activated protein kinase, heat shock factor 1 ( HSF1 ), and nutrient-deprivation autophagy factor-1 ( NAF-1 ) (69) ( Figure 2 )

Molecules 19 (12), 2009120112 (2014)

In prior clinical trials, CRWP was able to raise glutathione levels in those with obstructive lung disease (40, 41)

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Furthermore, FSP1 is highly expressed in various cancer cells and its expression has been linked to poor prognosis (18,19)