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Notably, ACSL1 is selectively upregulated in U-CLL cells and represents a targetable metabolic enhancer of ferroptosis sensitivity, as shown in vivo

5 Hypoxic stress at high altitude can trigger a series of physiological responses across multiple organ systems, especially the brain (increased cerebral blood flow), pulmonary system (increased ventilation and pulmonary vascular remodeling), cardiovascular system (increased heart rate, cardiac output, and systemic blood pressure), renal system (increased bicarbonate excretion and erythropoietin (EPO) secretion), and hematologic system (increased red cell mass, hemoglobin (Hb) concentration, and hematocrit (Hct)), thereby increasing oxygen content and delivery in the body

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While they show potential in enhancing therapeutic outcomes in cancer and other oxidative stress-related pathologies, challenges still remain like variable efficacy, lack of the standardization of doses, toxicity linked with high doses, etc

Synthetic lethal combination targeting BET uncovered intrinsic susceptibility of TNBC to ferroptosis