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Targeted inhibition of the Xc /GSH/GPX4 system axis effectively induces ferroptosis [79]

It enhances growth hormone receptor expression and several pathways involved in cell growth and angiogenesis, and increases nitric oxide production by upregulating nitric oxide synthase (NOS) gene and protein expression

Later, TRPM2 activator role of oxidative stress from ADPR was reported in transfected cells by single channel patch-clamp experiments (Naziroglu and Lckhoff, 2008)

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