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Mechanistically, the inflammatory response after heart transplantation is mediated by TLR4/TRIF signaling pathway, which facilitates neutrophil migration to coronary endothelial cells, resulting in ferroptosis in the transplant endothelial cells

This stress heightens cellular susceptibility to lipid peroxidation, ultimately promoting the execution of ferroptosis (67)

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Then, we investigated whether or not these genotypes affected GSH levels in the posterior medial frontal cortex