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The increased flux of acetyl-CoA will then build up the levels of Krebs intermediates including fumarate and succinate (also inhibitors of prolyl hydroxylase), especially if the biotin-dependent pyruvate carboxylase cannot sustain the production of oxaloacetate, the most limiting substrate of the cycle

Current research has found high concentration supplementation, at 180 mcg/day, results in improved clinical results compared to lower-dose supplementation.*

Hence, we have limited flexibility in establishing the performance threshold since we are restricted to the data we have available from the prior period

Both have their place in the right context, and sometimes the best protocol draws on both

This pathway is linked with various ailments including respiratory diseases, inflammatory diseases, auto immune diseases, cancer and diabetes