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Here, we summarized the role of the vicious cycle of mitochondrial damage and ROS accumulation in RA and introduced potential drugs that target mitochondria or scavenge ROS for RA treatment, with the aim of providing some help for the clinical treatment of RA ( Table 1 )

G., Neumann, J

In their 2014 lung cancer study they also found that antioxidant supplements caused lung tumor cells to turn off the activity of a well-known cancer-suppressing gene called p53

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Conclusion: Hepatocyte II-spectrin knockdown decreased PKM2 expression in Kupffer cells, suppressing pro-inflammatory cytokine production, and blocking NASH-associated HCC