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Description

In particular, astrocyte buffering is responsible for this process (78)

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When compared to experimental and clinical data on the accumulation of the byproducts of acetaminophen metabolism, APAP-S, APAP-G, and NAPQI-GSH, in the plasma and in the urine of humans, the model gives accurate predictions (see Figures 2, 3, 4 and 5)

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4 2 -nAChRs, increasing their spontaneous discharge frequency and thereby augmenting inhibitory inputs to dopaminergic neurons

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Metabolic syndrome and insulin resistance as risk factors for development of chronic kidney disease and rapid decline in renal function in elderly

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GBCA are gadolinium complexes, and the molecular structure of each GBCA determines its stability

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