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For R cells, the basal phosphorylation of NF-B showed an increasing tendency of nearly 17% (p = 0.1380), while a significant decrease was noted in the basal levels of phosphorylated ERK1/2 (near 50% decrease, p = 0.0077), compared to S cells (Fig

TNF-mediated JNK activation is believed to be mediated by ROS (superoxide radical anion and peroxyl radicals) because their specific scavengers have been shown to inhibit the activation of JNK

However, at pharmacologic or supraphysiological doses, it reduces transition metal ions such as Fe 3+ to Fe 2+ or Cu 2+ to Cu + , facilitating Fenton-like reactions that produce hydrogen peroxide (HO) and hydroxyl radicals in situ (82, 83)