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The pathophysiology of endothelial dysfunction is complex, in that it is based on endothelial nitric oxide synthase uncoupling and endothelial activation following stimulation by various inflammatory mediators (molecular patterns, oxidized lipoproteins, and cytokines) (46)

Compared to mouse macrophages, the primary bovine PBMC used in this study constituted a heterogenous cell population, comprising a monocyte population of approximately 10% as shown by hematological analyses (11, 17)

[9] Eder, K., Felgner, J., Becker, K., & Kluge, H

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The study was performed according to The Code of Ethics of the World Medical Association (Declaration of Helsinki) for studies involving humans