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To suppress disulfidptosis, two protein repair systems, the Trx system and the glutaredoxin (Grx) system, balance the conversion of thiol groups and disulfide bonds

This section will explore the molecular mechanisms involved in neurotransmitter dysregulation due to chronic ethanol consumption and how these alterations contribute to the pathophysiology of depression

Minutolo A, Petrone V, Fanelli M et al (2023) Thymosin alpha 1 restores the immune homeostasis in lymphocytes during Post-Acute sequelae of SARS-CoV-2 infection

Simultaneous pharmacologic inhibition of yes-associated protein 1 and glutaminase 1 via inhaled poly(lactic-co-glycolic) acidencapsulated microparticles improves pulmonary hypertension

Figure 2 2.1 Modulation of innate immune cell populations by bile acids 2.1.1 Macrophages: polarization, phagocytosis, and cytokine production Macrophages are key targets of bile acids (BAs) in the TIME, given their immune plasticity and roles in inflammation and tumor progression ( Macrophage BA responses are mediated by receptors such as farnesoid X receptor (FXR), Takeda G protein-coupled receptor 5 (TGR5), vitamin D receptor (VDR), liver X receptor alpha (LXR), and peroxisome proliferator-activated receptor gamma (PPAR) ( However, BA effects are context-dependent