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In the MYO among upregulated DEGs detected in MYO-1, two were related to mechanisms associated with cellular/endoplasmic reticulum stress: Scd2, described above, and Hspb1 (Heat Shock Protein Family B (Small) Member 1) involved in AMPK/HSPB1-mediated inhibition of inflammation and ER stress in skeletal muscle under hyperlipidemic conditions 46

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