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Liver fibrosis progression is governed by bidirectional crosstalk between T cells and hepatic stellate cells (HSCs)

Glutathione transferase (GST) forms a second phase of detoxification, catalyzing deactivation of a number of harmful compounds, requiring reduced glutathione as a cofactor, which is irreversibly consumed during GSH conjugation

The decrease in glutathione in the body causes waste substances called free radicals to damage cells

Anticatabolic effects may occur by improving nitrogen balance either via increased protein synthesis or reduced protein degradation

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