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Serum samples with identical apoA-I and HDL-C levels were found to have differing cholesterol acceptance capacities, mostly due to pre-beta HDL levels, which contributed to altered ABCA1-mediated cholesterol efflux (759)

We further validated the upregulation of TFRC/CD71 in CLL cells after in vitro and in vivo ibrutinib treatment (Fig

Collagen type XI alpha 1 (COL11A1) is a novel biomarker that is associated with chemotherapy resistance in a variety of cancers [62, 63]

The proposed mechanisms include upregulation of beta-3 adrenergic receptors in adipose tissue, activation of the cAMP-PKA lipolytic cascade through enhanced catecholamine sensitivity, direct or indirect inhibition of acetyl-CoA carboxylase (ACC) reducing de novo lipogenesis by approximately 43-57% in vitro, and activation of hormone-sensitive lipase (HSL) with increased glycerol release