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This means it has 10 times higher energy and glucose consumption than the other tissues and a high dependence on mitochondrial energy production, responsible for the increase of ROS (Correia et al., 2010

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42 This can be due to mutations in oncogenes or tumor suppressor genes and, as described below, can significantly influence sensitivity of tumors to anti-tumor immunity and therapeutic ferroptosis

*Correspondence: Lin Wu, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers