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The interaction of UCB with microglia initially affects defensive processes involved with the expression nuclear factor kappa B (NF-B) and of mitogen-activated protein kinases (MAPKs) as well as enhanced phagocytosis and the subsequent generation of pro-inflammatory cytokines

10.1007/978-1-4471-2906-6_24 Intermittent Hypoxia and Human Diseases 41 ManukhinaE

Limited immunogenicity of human induced pluripotent stem cell-derived cartilages

Key Facts Molecular Cause of Regeneration Loss: In aged muscle, HGF is chemically modified by nitration at tyrosine sites Y198 and Y250, preventing it from docking to c-met receptors on resident satellite cells to trigger muscle repair