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Description

An ALYREF-MYCN coactivator complex drives neuroblastoma tumorigenesis through effects on USP3 and MYCN stability

doi: 10.26355/eurrev_202311_34343 48 KadhimAPDNQ

(2025) 188:4530-4548e4525

With the current absence of technologies to rectify the actual mutations that underlie mitochondrial diseases, medical science can only address the resulting oxidative stress as a potential therapeutic option

It helps regulate cellular redox balance, participates in the metabolism of certain drugs and environmental compounds, and supports normal immune and mitochondrial function