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iNOS-derived NO is classically recognized as a critical effector for microbial killing in murine macrophages, and capable of forming cytotoxic peroxynitrite through rapid reaction with superoxide (40, 41)

It has also been reported that this polyphenol positively regulates miRNA-15 and 16, which results in the induction of apoptosis in breast cancer cells (Chang and Yu, 2016)

We found that monochlorobimane responded in a dose-dependent, age and structure specific manner to both chemicals, indicating its broad application potential as an unbiased method to assess glutathione interruptions arising from developmental chemical exposures in live embryos