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PER1 affects cancer growth and causes an increased rate of ionizing radiation-induced cell death, but it is unclear whether its lack of expression precedes and contributes to cancer development, or is the only source of cancer

This suggests that Epitalon may not just restore telomere length through enzymatic activation, but it may actually reprogram the transcriptional readiness of cells, shifting their expression profiles toward a more youthful state

This holds particular promise for: Older adults facing chronic low-grade inflammation Immunocompromised individuals Age-related decline in vaccine efficacy Oncology patients recovering from chemo- or radiotherapy-induced immune depletion In the next section, we turn to the brain by examining Epitalons neuroprotective potential and early findings on cognitive health and neurodegeneration

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