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Notably, the hepsin / mice exhibited a more pronounced increase in ROS levels after thioacetamide treatment compared with wild-type mice, which aligned with our hypothesis that hepsin / mice experience heightened ROS stress in the early stages of APAP exposure due to their elevated expression of GJ proteins (Fig

MORG1 limits mTORC1 signaling by inhibiting Rag GTPases

Regulation of nucleotide metabolism by mutant p53 contributes to its gain-of-function activities