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Thus, cardiolipin is highly sensitive to ROS-induced oxidative damage [145]

Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury

Overexpression of TFEB leads to degradation of bulk autophagic substrates such as damaged mitochondria (Nezich et al., 2015), long-lived protein aggregates (Martini-Stoica et al., 2016), and lipid droplets (Settembre et al., 2013), indicating the crucial role of TFEB in organelle-specific autophagy, such as mitophagy and lipophagy (Settembre et al., 2013

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