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Given that PI3K/AKT signaling regulates ELF3 expression, these findings support a model in which HER2HER3-mediated PI3K/AKT activity uniquely sustains the ELF3KRAS transcriptional program in KRAS G13D cells

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It suppresses ECM degradation, counteracts RSL3 mediated inhibition of GPX4, increases intracellular GSH levels and the GSH/GSSG ratio, reduces MDA content, and inhibits activation of the MAPK/NF-KB pathways, thereby protecting chondrocytes from ferroptosis (156)