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Further studies will be needed to verify the contribution of this mechanism to the DADA2 phenotype and to evaluate its potential as a therapeutic target

Figure 1 Through the interaction of polyphenols with gut microbiota, intestinal barrier function can be enhanced, and simultaneously, the richness and activity of the gut microbiota increase (41, 42)

In the absence of GPX4, the balance between the formation and reduction of lipid hydroperoxides (LOOH) is disrupted, leading to an imbalance in the cellular redox state

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Although direct evidence is still needed, pharmacological modulation of autonomic balance (e.g., -adrenergic blockade), dietary manipulation of microbial substrates, or short-term probiotic or postbiotic interventions might reveal ENS-microbiota interactions