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Louis, Alexis St

Those with certain genetic traits might be more sensitive

We also observed that this protection against ferroptosis can be abolished by the chemical inhibition of the biosynthesis of either CoA (PANKi) or GSH (BSO and DEM), indicating the important role of both ferroptosis-protecting branches

Disclosures: Francisco Caballero-Camino: Nothing to Disclose, Irune Lasa: Nothing to Disclose, Ainhoa Lapitz: Nothing to Disclose, Xia Wu: CymaBay Therapeutics: Employee, Pedro Rodrigues: Nothing to Disclose, Noelia Pastor-Toyos: Nothing to Disclose, Laura Izquierdo-Sanchez: Nothing to Disclose, Jiangao Song: Nothing to Disclose, Edward Cable: CymaBay Therapetics: Employee, Jeffrey Johnson: CymaBay Therapeutics: Consultant, Yun-Jung Choi: Nothing to Disclose, Charles McWherter: Nothing to Disclose, Jesus Banales: Nothing to Disclose 2077 REGULATION OF HEPATIC MULTIDRUG RESISTANCE-ASSOCIATED PROTEIN 3/ATP-BINDING CASSETTE SUBFAMILY C MEMBER 3 (MRP3/ABCC3) BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR (PPAR): A NOVEL MECHANISM TO REDUCE BILE ACID INJURY DURING CHOLESTASIS Colleen Hayes 1 Gina Gallucci 1 James Boyer 2 David Assis 2 Nisanne Ghonem 1 , 1 University of Rhode Island, 2 Yale School of Medicine, Liver Center Background: Cholestatic liver diseases are characterized by the accumulation of toxic bile acids

For example, soaking, autoclaving, and boiling cause irreversible denaturation of lectins