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Among the ACSL family members, ACSL4 preferentially catalyzes the formation of arachidonoyl-CoA (AA-CoA) by inserting coenzyme A (CoA) into arachidonic acid (AA), also called 5,8,11,14-eicosatetraenoic acid (20:4n-6, -6)

Sell C, Ptasznik A, Chang CD, Swantek J, Cristofalo VJ, Baserga R: IGF-1 receptor levels and the proliferation of young and senescent human fibroblasts

ATF4-mediated upregulation of SESN2 and DDIT4 inhibits mTORC1 activity in response to sustained deprivation of amino acids such as leucine, serving as a negative feedback signal to suppress mTORC1 signaling and downregulate amino acid transporters (Xu et al., 2020)

Acknowledgements We are grateful to CAPES, FAPESP and GlaxoSmithKline for their financial support

When evaluating side effects, the following frequencies are used as a basis: Very common: more than 1 in 10 people Common: less than 1 in 10, but more than 1 in 100 patients Uncommon: less than 1 in 100, but more than 1 in 1000 patients Rare: less than 1 in 1,000 but more than 1 in 10,000 patients Very rare: less than 1 in 10,000 patients treated, or unknown