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Data are presented as median with interquartile ranges

J Exp Med 218(6):e20210518

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10.1038/s41598-018-31877-6 Summary Keywords atherosclerosis, cell metabolism, glycolysis, fatty acids, diabetes, comorbidity Citation Dai Y, Junho CVC, Schieren L, Wollenhaupt J, Sluimer JC, van der Vorst EPC and Noels H (2024) Cellular metabolism changes in atherosclerosis and the impact of comorbidities

Research has primarily focused on its potential role in: Tendon, ligament, and muscle regeneration Supporting fibroblast migration Angiogenesis Regulation of vascular responses Protection of gastrointestinal tissues Recovery processes following experimentally induced injuries Experimental studies using cell cultures and animal models suggest that BPC-157 may promote fibroblast proliferation, stimulate collagen synthesis, and influence signaling pathways involved in tissue regeneration, including the FAK-paxillin and VEGFR2 signaling pathways