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[1] For glycolysis to continue, NADH must be reoxidized to NAD +

FSTL1 promotes liver fibrosis by reprogramming macrophage function through modulating the intracellular function of PKM2

In a mouse model of FA-induced AKI, Fer-1 significantly restored renal function, but apoptosis inhibitors were not effective, suggesting a dominant role for ferroptosis in FA-induced AKI [136, 163]