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This NO production is central to many biological functions, including the regulation of blood pressure, oxygen delivery and endothelial (blood vessel lining) health[1]

OCTN2 ) mutations, typically manifests in children aged 17 years, with a frequency of 15 per 10.000 and causes metabolic derangements including hypoketotic hypoglycemia, hepatic steatosis, myopathy, and potentially sudden death due to cardiomyopathy [4, 6]

3942 Results and discussion The first part of the results and discussion will focus on identifying unknown patients at risk for hyperammonemia

DOI: 10.1016/j.jad.2018.04.111

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