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Via regulation of p53 deacetylation and modulation of autophagy, SIRT1 is implicated in cellular response to caloric restriction and lifespan extension

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Its strong antioxidant properties shield skin cells from oxidative stress, which can worsen inflammation, by helping neutralise free radicals

The face contains subcutaneous fat pads that support skin structure and expression

Furthermore, SYNCRIP depletion resulted in a marked decrease in intracellular NAD + levels, a crucial cofactor required for the enzymatic activity of the NAD + -dependent deacetylase SIRT1 (Figure S4F-H) [27]