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The initiation of an inflammatory response results in IB being phosphorylated by IB kinase, thus becoming a target for ubiquitination and subsequent degradation

long-term human data is limited

At the same time, direct evidence shows that fibroblast migration is modulated through extracellular vesicles derived from macrophages [153], as the vesicles carry a rich cargo of growth factors, cytokines, and microRNAs that can alter the inflammatory milieu, promote angiogenesis, and directly or indirectly stimulate fibroblast proliferation and activation [151,153,154]