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In a study conducted by Hu and colleagues [219], it was observed that CNS damage occurred shortly after radiation exposure due to the activation of microglia, altered morphology, activation of injured nerve transcription factors, and production of inflammatory mediators

Homology modeling The homology model was made to establish secondary chimeras and to visualize the effects of mutations

Protects gut lining and other tissues from damage, reduces inflammation, and supports the bodys natural defensive and regenerative mechanisms

ChaC1 is highly expressed in gastric cancer associated with H

On the other hand, certain SCFAs under specific conditions might contribute to pathology: for example, a recent study found that butyrate and propionate can activate the NLRP3 inflammasome in human macrophages under inflammatory stress, suggesting a potential pro-inflammatory role in some contexts [78, 79]