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Its bile-salt structure helps reduce bile acid toxicity within liver cells by promoting more fluid bile and protecting the mitochondria from stress

Finally, we investigated the molecular mechanism by which VRK2 promotes GSH biosynthesis and found that VRK2 drives the trafficking of SLC7A11 through phosphorylating Sec24C

Notably, the activity of the Hif1 heterodimer is mostly controlled at the level of stability and expression of Hif1a and Hif2a subunits [101]

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