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FIGURE 6 Insertional Inactivation of copB Increases the Sensitivity of CAUH2 to H 2 O 2 In order to investigate the role of CopB in H 2 O 2 stress, a copB mutant, designated CAUH2copB, was constructed by homologous recombination and subjected to challenge assays

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The other hypothesis suggested that a K + channel protein is the oxygen sensor and inhibition of this channel and the ensuing depolarization is the initial event in transduction, as indicated by Peers and Kemp [1]

Second, a substantial portion of evidence comes from animal models, which may not fully reflect human physiological responses [17]