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It has been reported that upregulation of -catenin expression can impede the proliferation of pathogenic bacteria, thereby mitigating weaning stress-induced intestinal inflammation and damage to the intestinal barrier (Tong et al., 2023)

Simultaneously in another study it has been suggested that higher levels of PD-L1 in EVs from fibroblasts upon TGF- stimulation could contribute to immune suppression and fibrogenesis [70, 72]

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Importantly, therapeutic strategies aimed at concurrently enhancing peroxisomal function and mitigating mitochondrial oxidative stress have demonstrated efficacy in ameliorating ferroptosis and improving lung function in experimental models, highlighting the promise of targeting this organellar network