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Mechanistically, SSBP1 interacted with p53 and promoted DNA-PK-dependent phosphorylation of p53 at S15 to facilitate the nuclear accumulation of p53, which ultimately inhibited the transcription of SLC7A11 during ferroptosis in high fructose-treated glomerular podocytes (Fig

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Figure 15 shows the occurrence frequency (in percentage) of angular differences throughout all of the experiments that fall within a given time range

Progress in the clinical use of bone marrow aspirate concentrate for knee osteoarthritis: an expert opinion

The modified hGH fragment doesnt have hGHs growth-promoting effects