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Loss of SLC25A11 resulted in increased mitochondrial and cytoplasmic lipid peroxidation leading to ferroptosis To unravel the metabolic reprogramming in the SLC25A11 cell model, we further investigated several events by metabolomic profiling

Basu S, Sengupta B, Paladhi PK

The NUB1 UBL domain, thus, localizes the NUB1FAT10 complex to the proteasome for FAT10 engagement and degradation, with no obvious allosteric effects, which also agrees with our structural data presented below

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The histological properties of nerve fibers were improved in the STI + CoQ10 group, including enhanced myelin sheath thickness and axon diameters