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The monothiol mechanism for the reduction of non-glutathione disulfides is controversial and at least three different nonexclusive mechanisms have been discussed to date (Fig

We further observed that pretreatment with C1-27 inhibited the transcriptional induction of pro-IL-1 in response to inflammatory stimulus such as the bacterial endotoxin, lipopolysachcharide

Neuronal FAM171A2 mediates alpha-synuclein fibril uptake and drives parkinson's disease

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Relationship to previous work Cell clusters are common in degenerated discs, especially those that are herniated 36 , and are associated with cell proliferation 12,37 and with progenitor cells 38