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This activation disrupts the equilibrium between pro-inflammatory and anti-inflammatory processes, particularly within brain tissues (Proneth and Conrad, 2019)

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CaMKK2 provides a potential link between metabolic dysfunction and bipolar disorder Abnormalities in brain and whole-body energy metabolism are some of the most consistent features of bipolar disorder [17]

and (B) VMAT2-mediated exchange transport, in which amphetamine enters vesicles while monoamines are transported into the cytoplasm