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doi: 10.1038/s41598-017-13257-8

47:W212W224

Therefore, the implication from this work is that a large portion of the S in normal cellular steady-state GSH pools is derived from Met via the Met-cycle and transsulfuration

Some have demonstrated a close relationship between GST M1 or T1 polymorphisms and the susceptibility to SNHL 6,22,23,24,25 , and others have reported conflicting results 8,10,16,26,27,28,29,30,31,32,33,34

Intestine-specific FTN-deficiency leads to marked increases in intestinal iron absorption and inducing iron overload, and an unidentified novel microbial metabolite regulates systemic FTN homeostasis (Das et al., 2020)