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On the other hand, p53 inhibits ferroptosis by inhibiting the activity of dipeptidyl peptidase 4 (DPP4) or inducing the expression of cyclin-dependent kinase inhibitor 1A (CDKN1A/p21) [77], both indicate the importance of p53 as a regulator of metabolism-related genes in ferroptosis (Fig

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This approach led to the development of Cfa N and Cfa C intein fragments that can perform protein trans splicing (PTS) at high temperatures and in the presence of chaotropes ( Npu DnaE fragments in both mammalian and bacterial cells