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TRPV4-induced phenotypes include RGC degeneration (133), reactive gliosis (27), glial swelling (113) and increased permeability of the blood-retina barrier (127, 134) whereas mice lacking the TRPV4 gene seem to be protected from some types of mechanical and nociceptive stress (130) and do not appear to develop glaucoma (51)

Autoinflammatory glomerulonephritis stems from innate immunity errors, resulting in dysregulated cytokine production or complement activation

Low levels can be measured in a blood test

This genotype-dependent efficacy is robustly supported by epidemiological and clinical evidence in colorectal cancer, particularly for individuals with combined GSTM1 and GSTT1 null genotypes

The evidence highlights the potential of redox profiling as an indicator of metabolic pathology and as a measure of treatment response